Header links
Intranet
Donate now
image/svg+xml Map
Contact us

Fernando Martínez Valle

Fernando Martínez Valle

Institutions of which they are part

Systemic Diseases
Vall Hebron Institut de Recerca
Fernando Martínez Valle

Fernando Martínez Valle

Fernando Martínez Valle

Institutions of which they are part

Systemic Diseases
Vall Hebron Institut de Recerca

Projects

Estudi de les alteracions causades per autoanticossos sobre la cèl·lula endotelial mitjantçant la tècnica de mRNA DIFFERENTIAL DISPLAY en malalts amb síndrome de Sneddon

IP: Miguel Vilardell Tarres
Collaborators: Fernando Martínez Valle
Funding agency: Agència Gestió Ajuts Universitaris i de Recerca
Funding:
Reference: 2001FI 00054
Duration: 01/01/2001 - 31/12/2004

Related news

Equip estudi autoanticòs esclerodèrmia

Vall d'Hebron discovers a new antibody for the diagnosis of patients with scleroderma

Researchers have used an innovative protocol to identify people with this autoimmune disease who have a higher risk of cancer.

Conveni Synlab i VHIR

SYNLAB and Vall d'Hebron sign collaboration agreement to promote medical research

It will allow the promotion of research on systemic autoimmune-based diseases that especially affect women of childbearing age and, therefore, can also appear during pregnancy.

Joan_Vivancos_Trombosis_SAF

Joan Vivancos 2022 award to an article from the VHIR Systemic Diseases group

The study showing how thrombocytopenia increases the risk of thrombosis in patients with antiphospholipid syndrome has been considered the best paper published in 2021.

See more

Related professionals

Elisa Merino Peña

Elisa Merino Peña

Read more
Eulalia Gine Cipres

Eulalia Gine Cipres

Headache and Neurological Pain
Read more
Pau Torrella Llauger

Pau Torrella Llauger

Read more
Anas Chaachou Charradi

Anas Chaachou Charradi

Read more

Subscribe to our newsletters and be part of the Campus life

We are a world-leading healthcare complex where healthcare, research, teaching and innovation go hand in hand.

This question is for testing whether or not you are a human visitor and to prevent automated spam submissions.