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Lydia Dux-Santoy Hurtado

Institutions of which they are part

Postdoctoral researcher
Cardiovascular Diseases
Vall Hebron Institut de Recerca

Lydia Dux-Santoy Hurtado

Institutions of which they are part

Postdoctoral researcher
Cardiovascular Diseases
Vall Hebron Institut de Recerca

Projects

Aneurisma aterosclerótico de aorta torácica descendente y abdominal: predictores biomecánicos, histológicos y hemodinámicos de dilatación aórtica y eventos adversos

IP: Jose Fernando Rodríguez Palomares
Collaborators: Sergi Bellmunt Montoya, Aroa Ruiz Muñoz, Lydia Dux-Santoy Hurtado, Ruperto Olivero Soldevila
Funding agency: Instituto de Salud Carlos III
Funding: 127050
Reference: PI19/01480
Duration: 01/01/2020 - 30/06/2024

CardioP3T: Nuevos biomarcadores de diagnóstico y pronóstico en patología cardíaca a partir de técnicas de imagen híbrida PET-RM

IP: Jose Fernando Rodríguez Palomares
Collaborators: Lydia Dux-Santoy Hurtado, Andrea Guala
Funding agency: Ministerio de Ciencia e Innovación-MICINN
Funding: 138065.8
Reference: RTC2019-007280-1
Duration: 01/01/2020 - 30/06/2024

Ministerio de Ciencia

ANEURISMAS DE AORTA ASCENDENTE. PROGRESION DE LA DILATACION: ETIOPATOGENIA Y NUEVOS BIOMARCADORES DE IMAGEN PARA PREDECIR SU EVOLUCION

IP: -
Collaborators: Jose Fernando Rodríguez Palomares, Rafael Rodríguez Lecoq, Gisela Teixido Tura, Aroa Ruiz Muñoz, Hug Cuellar Calabria, Lydia Dux-Santoy Hurtado, Andrea Guala
Funding agency: Instituto de Salud Carlos III
Funding: 134310
Reference: PI17/00381
Duration: 01/01/2018 - 30/06/2022

Related news

Researchers incorporate advanced cardiovascular prediction algorithms into cardiac imaging software, enabling the automatic calculation of the risk of serious complications during the routine analysis of cardiac studies.

The event brought together experts from the Vall d’Hebron Institute of Research (VHIR) and the Institute of Geosciences of Barcelona to analyze how environmental factors and the climate crisis impact population health.

The accumulation of metabolic products within cardiac cells alters their function and accelerates the deterioration of heart tissue with age.

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