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Nicolás Miguel Fissolo

I am biochemist and PhD in human biology. Since 2008 I work as a researcher at the clinical neuroimmunology - Multiple Sclerosis Center of Catalonia (Cemcat) research group. My activity is based on the study of multiple sclerosis, mainly in the identification of molecular biomarkers associated to different aspects of the disease.

Institutions of which they are part

Main researcher
Clinical Neuroimmunology
Vall Hebron Institut de Recerca
Investigador/a postdoctoral
Multiple Sclerosis Centre of Catalonia

Nicolás Miguel Fissolo

Institutions of which they are part

Main researcher
Clinical Neuroimmunology
Vall Hebron Institut de Recerca
Investigador/a postdoctoral
Multiple Sclerosis Centre of Catalonia

I am biochemist and PhD in human biology. Since 2008 I work as a researcher at the clinical neuroimmunology - Multiple Sclerosis Center of Catalonia (Cemcat) research group. My activity is based on the study of multiple sclerosis, mainly in the identification of molecular biomarkers associated to different aspects of the disease.

I graduated as a biochemist in 1999 from the University of Córdoba (Argentina). In 2005 I obtained a PhD in human biology from the University of Ulm (Germany) for my work in the DNA vaccine field. Afterwards, I carried out a 3-year postdoctoral stay at the Hertie Institut for Brain Research at the University of Tübingen (Germany) focused on the study of antigenic presentation in multiple sclerosis. In 2008 I joined the research group of the clinical neuroimmunology - Cemcat directed by Dr. Xavier Montalban where I have been working until now.

Since 2010 I have been working as principal investigator of several projects focused on the identification of new molecular biomarkers that allow the characterization of the different aspects of the disease, among which are included prognostic biomarkers in the initial phases of the disease, biomarkers of response to treatment, and biomarkers of disease activity.

Projects

Immunogenicity: assesing the clinical relevance and risk minimization of antibodies to biopharmaceuticals. IMI Grant Agreement nº 115303

IP: Manuel Comabella Lopez
Collaborators: Carmen Tur Gomez, Nicolás Miguel Fissolo
Funding agency: The Innovative Medicines Initiative
Funding: 464328
Reference: ABIRISK_IMI2010
Duration: 01/03/2012 - 27/02/2018

Vacunas de ADN en Esclerosis Múltiple

IP: Nicolás Miguel Fissolo
Collaborators: -
Funding agency: Instituto de Salud Carlos III
Funding: 62315
Reference: PI10/01654
Duration: 01/01/2011 - 31/12/2014

Unitat de Neuroimmunologia Clínica (UNiC)

IP: Unitat de Neuroimmunologia Clínica (UNiC)
Collaborators: Susana Otero Romero, Carlos Nos Llopis, Unitat de Neuroimmunologia Clínica (UNiC), Jaume Sastre Garriga, Jordi Rio Izquierdo, Ingrid Galán Cartaña, Rosalia Horno Ocaña, Manuel Comabella Lopez, Carmen Tur Gomez, Unitat de Neuroimmunologia Clínica (UNiC), Herena Eixarch Ahufinger, Montserrat Moreno Sánchez, Unitat de Neuroimmunologia Clínica (UNiC), M Jesus Arevalo Navines, Carmen Espejo Ruiz, Angela Vidal Jordana, Joaquin Castillo Justribo, Mireia Castillo Juarez, Nicolás Miguel Fissolo , Georgina Arrambide García
Funding agency: Agència Gestió Ajuts Universitaris i de Recerca
Funding: 43680
Reference: 2009 SGR 793
Duration: 01/01/2010 - 30/04/2014

Vacunas de ADN en esclerosis múltiple.

IP: Vacunas de ADN en esclerosis múltiple.
Collaborators: Nicolás Miguel Fissolo
Funding agency: Fundació Institut de Recerca HUVH
Funding: 90000
Reference: POST/IR-HUVH/1/2008
Duration: 01/01/2009 - 31/12/2011

Related news

An international study led by researchers from Vall d’Hebron shows that simple tests that patients can perform themselves help detect changes in disability that may occur even in the absence of disease relapses.

A Vall d’Hebron study, carried out at Cemcat, shows that the risk of inflammatory activity increases after discontinuing treatment, but is considerably reduced in people over 60 years of age.

The project will analyse the functional activation of the NLRP3 inflammasome to advance the early identification of patients at risk of insufficient response to S1P receptor-modulating therapies.

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