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Anna Meseguer Navarro

Institutions of which they are part

Head of group
Kidney Physiopathology
Vall Hebron Institut de Recerca

Anna Meseguer Navarro

Institutions of which they are part

Head of group
Kidney Physiopathology
Vall Hebron Institut de Recerca

Projects

Mecanismos de Neuroprotección mediada por Receptores de Estrógenos de Membrana

IP: Anna Meseguer Navarro
Collaborators: -
Funding agency: Ministerio de Ciencia e Innovación-MICINN
Funding: 31350
Reference: JCI-2005-1469-1
Duration: 01/11/2006 - 30/11/2008

Ministerio de Ciencia

Mecanismos de protección en la toxicidad renal producida por ciclosporina A(CsA): Función de las ciclofilinas (CypA/CypB) y de la Kidney androgen-regulated protein (KAP)

IP: Anna Meseguer Navarro
Collaborators: Joan López Hellin
Funding agency: Ministerio de Ciencia e Innovación-MICINN
Funding: 173740
Reference: SAF2005-05167
Duration: 15/10/2005 - 14/10/2008

Ministerio de Ciencia

Patologia cel·lular

IP: Anna Meseguer Navarro
Collaborators: Joan López Hellin, Jesus Castro Marrero, Elisa Llurba Olivé
Funding agency: Agència Gestió Ajuts Universitaris i de Recerca
Funding:
Reference: 2005SGR 01000
Duration: 01/01/2006 - 31/12/2008

Mecanismes moleculars implicats en la nefroxicitat produïda per Ciclospirina A (CsA) i FK506 en el túbul proximal renal

IP: Anna Meseguer Navarro
Collaborators: -
Funding agency: Agència Gestió Ajuts Universitaris i de Recerca
Funding:
Reference: 2005FI 00195
Duration: 01/01/2005 - 31/12/2008

Related news

The communication is part of a study aimed at identifying the mechanisms of progression of Familial Hypomagnesemia with Hypercalciuria and Nephrocalcinosis, a rare disease that affects the kidneys.

"Patients with familial hypomagnesemia with hypercalciuria and nephrocalcinosis present miRNA profiles in urinary extracellular vesicles associated with disease progression" was the awarded work.

Researchers at the VHIR have carried out a study showing that the ClC-5 protein regulates collagen levels through the β-catenin pathway and lysosomal degradation.

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