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Anna Meseguer Navarro

Institutions of which they are part

Head of group
Kidney Physiopathology
Vall Hebron Institut de Recerca

Anna Meseguer Navarro

Institutions of which they are part

Head of group
Kidney Physiopathology
Vall Hebron Institut de Recerca

Projects

Implicación de la ciclofilina B (CYBP) y de las proteínas que con ella interaccionan, en el daño renal agudo

IP: Anna Meseguer Navarro
Collaborators: Joan López Hellin, Thais Cuadros Arasa
Funding agency: Fundación Renal Iñigo Alvarez de Toledo
Funding: 18000
Reference: FRIAT_01_2007
Duration: 01/01/2008 - 31/12/2009

Mecanismos moleculares implicados en la nefrotoxicidad producida por Ciclosporina A (CsA) y FK506 en el túbulo proximal renal.

IP: Anna Meseguer Navarro
Collaborators: Joan López Hellin
Funding agency: Instituto de Salud Carlos III
Funding: 154675
Reference: PI020829
Duration: 06/11/2002 - 06/11/2005

Implicacions del receptor del virus de l'hepatitis A humà (hHAVcr-1) en el desenvolupament i la progressió del carcinoma renal. Valor com a marcador diagnòstic i pronòstic en els carcinomes de bufeta i renals

IP: Anna Meseguer Navarro
Collaborators: Santiago Ramon y Cajal Agüeras, Joan Morote Robles, Enric Trilla Herrera, Thais Cuadros Arasa
Funding agency: Fundació La Marató de TV3
Funding: 204625
Reference: TV3/052410
Duration: 01/01/2006 - 30/04/2010

Las ciclofilinas en el fracaso renal agudo tóxico. Identificación de biomarcadores pronósticos y de posibles dianas terapéuticas.

IP: Anna Meseguer Navarro
Collaborators: Joan López Hellin
Funding agency: Fundación Invest. Médica Mutua Madrileña
Funding: 38200
Reference: FMMA/01/2005
Duration: 11/09/2006 - 10/09/2009

Related news

"Patients with familial hypomagnesemia with hypercalciuria and nephrocalcinosis present miRNA profiles in urinary extracellular vesicles associated with disease progression" was the awarded work.

Researchers at the VHIR have carried out a study showing that the ClC-5 protein regulates collagen levels through the β-catenin pathway and lysosomal degradation.

The research will perform a functional analysis of phenotype-modifying genetic variants in patients affected by familial hypomagnesaemia with hypercalciuria and nephrocalcinosis (HFHNC).

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