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Elisabet Miro Casas

Institutions of which they are part

Research technician
Cardiovascular Diseases
Vall Hebron Institut de Recerca

Elisabet Miro Casas

Institutions of which they are part

Research technician
Cardiovascular Diseases
Vall Hebron Institut de Recerca

Projects

Inhibición del intercambiador sodio/calcio mitocondrial, NCLX, como estrategia para reducir el daño por isquemia-reperfusión durante un infarto de miocardio

IP: Antonio Rodríguez Sinovas
Collaborators: Elisabet Miro Casas, Gino Freddy Sena Ganse
Funding agency: FUNDACIÓN ESPAÑOLA DE ARTERIOSCLEROSIS
Funding: 15000
Reference: FEA/RODRIGUEZ/2024
Duration: 01/01/2025 - 31/12/2027

Inhibición del intercanviador sodio/calcio mitocondrial, NCLX, como estrategia terapéutica para reducir el daño miocárdico por isquemia-reperfusión durante el infarto de miocardio

IP: Antonio Rodríguez Sinovas
Collaborators: Laia Yañez Bisbe, Elisabet Miro Casas, Marisol Ruiz Meana, Laura Rubio Unguetti, Gino Freddy Sena Ganse
Funding agency: Fundación Eugenio Rodríguez Pascual
Funding: 25000
Reference: FERP/2024/RODRIGUEZ
Duration: 15/01/2025 - 14/01/2027

Role of amino acid metabolism reprogramming and calpain deregulation in fibroblasts activation and diffuse myocardial fibrosis in heart failure

IP: Javier Inserte Igual
Collaborators: José Antonio Barrabés Riu, David Aluja Gonzalez, Juan Garrido Oliver, Elisabet Miro Casas, Sara Delgado Tomás
Funding agency: Instituto de Salud Carlos III
Funding: 152500
Reference: PI23/00068
Duration: 01/01/2024 - 31/12/2026

Daño mitocondrial por estrés dicarbonílico como impulsor de la senescencia: papel de los senolíticos en la protección del corazón durante el envejecimiento.

IP: Marisol Ruiz Meana
Collaborators: Rafael Rodríguez Lecoq, David Aluja Gonzalez, Elisabet Miro Casas
Funding agency: Instituto de Salud Carlos III
Funding: 159720
Reference: PI22/00513
Duration: 01/01/2023 - 31/12/2025

Related news

The accumulation of metabolic products within cardiac cells alters their function and accelerates the deterioration of heart tissue with age.

A study led by the Vall d'Hebron Research Institute (VHIR) demonstrates the causal role of air pollution in ventricular arrhythmias and identifies a possible therapeutic strategy based on antioxidants.

The grants, amounting to more than €533,000 for VHIR, will allow clinical duties to be partially released over three years to promote strategic research projects in obesity, respiratory diseases, aortic pathology and metabolic fatty liver disease.

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