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Julieta Torchia

Institutions of which they are part

Predoctoral researcher
Kidney Physiopathology
Vall Hebron Institut de Recerca

Julieta Torchia

Institutions of which they are part

Predoctoral researcher
Kidney Physiopathology
Vall Hebron Institut de Recerca

Projects

Contribución de la microbiota intestinal en la progresión del daño renal de pacientes con hipomagnesemia familiar con hipercalciuria y nefrocalcinosis

IP: Gema Ariceta Iraola
Collaborators: Cristina Martínez Martínez, Julieta Torchia Pruzzo
Funding agency: AIRG - ESPAÑA
Funding: 10000
Reference: CGE/ARICETA/2023
Duration: 01/01/2024 - 01/01/2025

Precision medicine approaches in idiopathic nephrotic syndrome: discovery of novel diagnostic/predictive biomarkers and therapeutic targets based on gut-microbiota-kidney crosstalk

IP: Cristina Martínez Martínez
Collaborators: Julieta Torchia Pruzzo
Funding agency: Instituto de Salud Carlos III
Funding: 146250
Reference: PI24/00547
Duration: 01/01/2025 - 31/12/2027

Biomarcadores, dianas y soluciones terapéuticas para mejorar el cuidado de pacientes afectados de Hipomagnesemia Familiar con Hipercalciuria y Nefrocalcinosis (HFHNC).

IP: Gema Ariceta Iraola
Collaborators: Hector Rios Duro, Mónica Durán Fernández, Gloria Mª Fraga Rodriguez, Julieta Torchia, Julieta Torchia Pruzzo
Funding agency: Instituto de Salud Carlos III
Funding: 202070
Reference: PI22/01946
Duration: 01/01/2023 - 31/12/2025

Papel del ClC-5 en la fibrosis renal. Identificación de posibles biomarcadores y dianas terapéuticas para la progresión de la enfermedad de Dent 1

IP: Gerard Cantero Recasens
Collaborators: Papel del ClC-5 en la fibrosis renal. Identificación de posibles biomarcadores y dianas terapéuticas para la progresión de la e, Hector Rios Duro, Mónica Durán Fernández, Gloria Mª Fraga Rodriguez, Julieta Torchia, Julieta Torchia Pruzzo, Andrea Casal Pardo
Funding agency: Instituto de Salud Carlos III
Funding: 123420
Reference: PI22/00741
Duration: 01/01/2023 - 31/12/2025

Related news

The team has discovered that a protein called SEC22B acts as a phenotype modifier and could be used as a potential urinary marker of the disease.

Funding has been obtained for 43 projects under the calls for Health R&D&I Projects, Health Technology Development, and Independent Clinical Research

The aim of the project is to establish kidney organoids derived from patients with familial hypomagnesaemia with hypercalciuria and nephrocalcinosis, which will be essential tools for studying the disease and testing new treatments.

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