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Cristina Martínez Martínez

Institutions of which they are part

Main researcher
Kidney Physiopathology
Vall Hebron Institut de Recerca

Cristina Martínez Martínez

Institutions of which they are part

Main researcher
Kidney Physiopathology
Vall Hebron Institut de Recerca

Projects

Unitat de Recerca del Sistema Digestiu

IP: Fernando Azpiroz Vidaur
Collaborators: Fco Javier Santos Vicente, Beatriz Lobo Alvarez, Natalia Borruel Sainz, María Antolín Mate, Mar Guilarte Clavero, Francesc Casellas Jordá, Carmen Alonso Cotoner, Carolina Malagelada Prats, Jorge Serra Pueyo, Cristina Martínez Martínez, Chaysavanh Manichanh
Funding agency: Agència Gestió Ajuts Universitaris i de Recerca
Funding: 50960
Reference: 2009 SGR 219
Duration: 01/01/2010 - 30/04/2014

Efecto de la estabilización prolongada del mastocito intestinal con cromoglicato disódico en la evolución clínica y la microinflamación de la mucosa intestinal en los pacientes con síndrome de intestino irritable tipo diarrea

IP: Fco Javier Santos Vicente
Collaborators: Beatriz Lobo Alvarez, Mar Guilarte Clavero, Inés de Torres Ramirez, Cristina Martínez Martínez
Funding agency: Instituto de Salud Carlos III
Funding: 168190
Reference: EC07/90148
Duration: 01/11/2007 - 30/12/2011

Señales anti-inflamatorias del ecosistema microbiano intestinal

IP: -
Collaborators: Natalia Borruel Sainz, María Antolín Mate, Cristina Martínez Martínez, Chaysavanh Manichanh
Funding agency: Ministerio de Ciencia e Innovación-MICINN
Funding: 169400
Reference: SAF2007-64411
Duration: 01/10/2007 - 24/10/2010

Ministerio de Ciencia

Efecto del estrés sobre la composición de la flora bacteriana y la respuesta inmune innata en el yeyuno humano

IP: Fco Javier Santos Vicente
Collaborators: Mar Guilarte Clavero, Carmen Alonso Cotoner, Cristina Martínez Martínez
Funding agency: Instituto de Salud Carlos III
Funding: 59500
Reference: PI051423
Duration: 01/01/2006 - 31/12/2008

Related news

The aim of the project is to establish kidney organoids derived from patients with familial hypomagnesaemia with hypercalciuria and nephrocalcinosis, which will be essential tools for studying the disease and testing new treatments.

The work identifies variants in genes such as NFU1 that, combined with the disease-causing mutation, can accelerate kidney deterioration.

The communication is part of a study aimed at identifying the mechanisms of progression of Familial Hypomagnesemia with Hypercalciuria and Nephrocalcinosis, a rare disease that affects the kidneys.

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