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Cristina Martínez Martínez

Institutions of which they are part

Main researcher
Kidney Physiopathology
Vall Hebron Institut de Recerca

Cristina Martínez Martínez

Institutions of which they are part

Main researcher
Kidney Physiopathology
Vall Hebron Institut de Recerca

Projects

Candidatura Juan de la Cierva-incorporación

IP: Fco Javier Santos Vicente
Collaborators: Cristina Martínez Martínez
Funding agency: Ministerio Economía, Industria y Competitividad
Funding: 64000
Reference: IJCI-2015-26099
Duration: 09/01/2017 - 08/01/2019

Identificación de antígenos inductores de la respuesta humoral y de mecanismos de regulación de la inflamación intestinal en el síndrome del intestino irritable. Desarrollo de un panel diagnóstico diferencial.

IP: -
Collaborators: Inés de Torres Ramirez, Identificación de antígenos inductores de la respuesta humoral y de mecanismos de regulación de la inflamación intestinal en el , Marc Pigrau Pastor, Cristina Martínez Martínez, Danila Guagnozzi
Funding agency: Instituto de Salud Carlos III
Funding: 183012.5
Reference: PI16/00583
Duration: 01/01/2017 - 30/06/2020

Desarrollo de un panel diagnóstico innovador para el Síndrome del Intestino Irritable basado en la expresión sistémica diferencial de miRNAs, asociado a manifestaciones clínicas y a patología intestinal

IP: Fco Javier Santos Vicente
Collaborators: Ana Maria Gonzalez Castro, Cristina Martínez Martínez
Funding agency: Instituto de Salud Carlos III
Funding: 171215
Reference: PI14/00994
Duration: 01/01/2015 - 30/04/2018

HNF4A EXPRESSION AND REGULATION IN THE JEJUNUM OF IBS-D PATIENTS

IP: Fernando Azpiroz Vidaur
Collaborators: Cristina Martínez Martínez
Funding agency: Agència Gestió Ajuts Universitaris i de Recerca
Funding: 40604.62
Reference: 2011 BP_A2 00002
Duration: 01/02/2015 - 31/01/2016

Related news

Funding has been obtained for 43 projects under the calls for Health R&D&I Projects, Health Technology Development, and Independent Clinical Research

The aim of the project is to establish kidney organoids derived from patients with familial hypomagnesaemia with hypercalciuria and nephrocalcinosis, which will be essential tools for studying the disease and testing new treatments.

The work identifies variants in genes such as NFU1 that, combined with the disease-causing mutation, can accelerate kidney deterioration.

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