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Cristina Martínez Martínez

Institutions of which they are part

Main researcher
Kidney Physiopathology
Vall Hebron Institut de Recerca

Cristina Martínez Martínez

Institutions of which they are part

Main researcher
Kidney Physiopathology
Vall Hebron Institut de Recerca

Projects

Contribución de la microbiota intestinal en la progresión del daño renal de pacientes con hipomagnesemia familiar con hipercalciuria y nefrocalcinosis

IP: Gema Ariceta Iraola
Collaborators: Cristina Martínez Martínez, Julieta Torchia
Funding agency: AIRG - ESPAÑA
Funding: 10000
Reference: CGE/ARICETA/2023
Duration: 01/01/2024 - 01/01/2025

Interacción entre huésped-microbiota y defectos en la integridad de la mucosa intestinal implicados en el origen del daño glomerular en el síndrome nefrótico idiopático

IP: Cristina Martínez Martínez
Collaborators: Alejandro Cruz Gual, Alvaro Domingo Madrid Aris, Ana Maria Gonzalez Castro
Funding agency: Instituto de Salud Carlos III
Funding: 38585.07
Reference: PI21/00757
Duration: 01/01/2022 - 30/06/2026

Precision medicine approaches in idiopathic nephrotic syndrome: discovery of novel diagnostic/predictive biomarkers and therapeutic targets based on gut-microbiota-kidney crosstalk

IP: Cristina Martínez Martínez
Collaborators: Julieta Torchia
Funding agency: Instituto de Salud Carlos III
Funding: 146250
Reference: PI24/00547
Duration: 01/01/2025 - 31/12/2027

Validación e implementación de un panel molecular para el diagnóstico positivo del síndrome del intestino irritable con diarrea y para el diagnóstico diferencial de la diarrea crónica

IP: Fco Javier Santos Vicente
Collaborators: Ana Maria Gonzalez Castro, Lara Aguilera Castro, Marc Pigrau Pastor, Cristina Martínez Martínez, Adoración Nieto Ruiz
Funding agency: Instituto de Salud Carlos III
Funding: 220220
Reference: PI17/01902
Duration: 01/01/2018 - 30/06/2022

Related news

The aim of the project is to establish kidney organoids derived from patients with familial hypomagnesaemia with hypercalciuria and nephrocalcinosis, which will be essential tools for studying the disease and testing new treatments.

The work identifies variants in genes such as NFU1 that, combined with the disease-causing mutation, can accelerate kidney deterioration.

The communication is part of a study aimed at identifying the mechanisms of progression of Familial Hypomagnesemia with Hypercalciuria and Nephrocalcinosis, a rare disease that affects the kidneys.

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